πŸ₯ Nursing Β· Med-Surg

Memory tricks for med-surg nursing

Cardiac, respiratory, renal, GI, endocrine, and neuro assessment β€” highest-yield med-surg topics.

πŸ₯ Med-Surg Nursing

Memory Tricks

Proven Mnemonics & Acronyms β€” fast to learn, hard to forget.

Cancer Warning Signs
CAUTION UP
Change Β· A lesion Β· Unusual bleeding Β· Thickening Β· Indigestion Β· Obvious skin changes Β· Nagging cough Β· Unexplained weight loss Β· Pernicious anemia
Early cancer warning signs β€” early detection saves lives
CAUTION UP helps nurses remember the key indicators of cancer. Early detection dramatically improves treatment outcomes β€” knowing these signs helps you advocate for your patients before symptoms worsen.
C
Change in bowel or bladder habits
A
A lesion or sore that will not heal
U
Unusual bleeding or discharge
T
Thickening or lump in breast or tissue
I
Indigestion or difficulty swallowing
O
Obvious change in wart or mole
N
Nagging cough or hoarseness
U
Unexplained weight loss or loss of appetite
P
Pernicious anemia β€” B12 deficiency
πŸ“– Full Lesson β†’
Heart Failure
Left-sided HF: pulmonary symptoms (SOB, crackles, pink frothy sputum). Right-sided HF: systemic edema (JVD, pitting edema, weight gain).
Heart Failure β€” Left vs Right
Telling left from right heart failure: lungs vs body β€” the most testable distinction
Left-sided HF (most common): left ventricle fails β†’ blood backs up into pulmonary circulation. Signs: dyspnea (especially at rest or lying flat β€” orthopnea), paroxysmal nocturnal dyspnea, crackles in lungs, pink frothy sputum (severe), decreased SpO2, S3 gallop. Right-sided HF: right ventricle fails β†’ backs up into systemic circulation. Signs: JVD (jugular vein distension), dependent pitting edema (ankles, sacrum), hepatomegaly, ascites, weight gain. Both: fatigue, decreased activity tolerance. Management: daily weights (report >2 lb gain in 1 day or >5 lb in 1 week), fluid restriction, low-sodium diet, elevate HOB, oxygen.
Left HF
Lungs β€” SOB, crackles, orthopnea
Right HF
Body β€” JVD, edema, weight gain
Monitor
Daily weight β€” >2 lb = call provider
Position
HOB elevated 30–45Β°
πŸ“– Full Lesson β†’
Coronary Arteries
RIGHT CAMP LEFT AC
Right Coronary Artery Β· Marginal Artery Β· Posterior Intraventricular Β· Left Anterior Descending Β· Circumflex Artery
I have a RIGHT to CAMP if you LEFT off the AC
Coronary arteries supply oxygenated blood to the heart muscle. Understanding their location is essential for cardiac nursing β€” knowing which artery is blocked tells you which part of the heart is at risk during an MI.
R
Right coronary artery
C
Marginal artery (branch of right)
A
Posterior intraventricular artery
M
Left coronary artery (main)
P
Anterior descending artery (LAD β€” widowmaker)
LEFT AC
Left Anterior descending + Circumflex artery
πŸ“– Full Lesson β†’
Heart Murmurs
hARD ASS MRS. MSD
Aortic Regurg=Diastolic Β· Aortic Stenosis=Systolic Β· Mitral Regurg=Systolic Β· Mitral Stenosis=Diastolic
Remember which phase you hear each heart murmur
Heart murmurs occur when turbulent blood flow creates abnormal sounds. Knowing which phase (systolic vs diastolic) a murmur occurs in is critical for identifying the underlying valve problem on NCLEX and in clinical practice.
hARD
Aortic Regurgitation = Diastolic murmur
ASS
Aortic Stenosis = Systolic murmur
MRS.
Mitral Regurgitation = Systolic murmur
MSD
Mitral Stenosis = Diastolic murmur
πŸ“– Full Lesson β†’
Cardiac Valves
Toilet Paper My Ass
Tricuspid Β· Pulmonic Β· Mitral Β· Aortic
Order of blood flow through the four heart valves
Blood flows through the heart valves in a specific sequence. Remembering this order is essential for understanding cardiac physiology, heart sounds, and valve disorders. The sequence follows blood from the right side to the left side of the heart.
T
Tricuspid β€” right atrium to right ventricle
P
Pulmonic β€” right ventricle to pulmonary artery
M
Mitral β€” left atrium to left ventricle
A
Aortic β€” left ventricle to aorta and body
πŸ“– Full Lesson β†’
Heart Sounds
APE To Man
Aortic Β· Pulmonic Β· Erb's point Β· Tricuspid Β· Mitral
Auscultation sites for heart sounds in order
APE To Man gives you the five auscultation sites in order. Aortic and Pulmonic are both in the 2nd intercostal space (2 words, 2nd space). Erb's point is in the 3rd. Tricuspid is in the 5th left sternal border. Mitral (sounds like "mid") is at the midclavicular line.
A
Aortic β€” 2nd intercostal space, right sternal border
P
Pulmonic β€” 2nd intercostal space, left sternal border
E
Erb's point β€” 3rd intercostal space, left sternal border
T
Tricuspid β€” 5th intercostal space, left sternal border
M
Mitral β€” 5th intercostal space, midclavicular line
πŸ“– Full Lesson β†’
Appendicitis
PAINS
Pain RLQ Β· Anorexia Β· Increased temp/WBC Β· Nausea Β· Signs (McBurney's, Psoas)
Classic assessment findings of appendicitis
Appendicitis is one of the most common surgical emergencies. PAINS helps you recognize it quickly. Pain typically starts periumbilical then migrates to the right lower quadrant. McBurney's point is 1/3 the distance from the anterior superior iliac spine to the navel.
P
Pain in right lower quadrant β€” rebound tenderness
A
Anorexia β€” loss of appetite
I
Increased temperature and WBC (15,000-20,000)
N
Nausea and vomiting
S
Signs β€” McBurney's point, Psoas sign
πŸ“– Full Lesson β†’
Hypercalcemia
GROANS MOANS BONES STONES OVERTONES
Constipation Β· Joint pain Β· Bone loss Β· Kidney stones Β· Psychiatric symptoms
Signs and symptoms of hypercalcemia β€” the rhymes make it stick
Hypercalcemia occurs when calcium levels exceed 10.5 mg/dL. Common causes: hyperparathyroidism, malignancy, prolonged immobility, excess vitamin D. The rhyming words make these symptoms impossible to forget β€” and they're high yield for NCLEX.
GROANS
Constipation β€” GI slowing
MOANS
Joint and bone pain
BONES
Loss of calcium from bones β€” osteoporosis
STONES
Kidney stones β€” hypercalciuria
OVERTONES
Psychiatric β€” confusion, depression, psychosis
πŸ“– Full Lesson β†’
Addison's Disease
STEROID
Sugar/Sodium low Β· Tired/muscle weakness Β· Electrolyte imbalance Β· Reproductive change Β· lOw BP Β· Increased pigmentation Β· Diarrhea/Depression
Assessment findings of Addison's Disease β€” low adrenal hormones
Addison's Disease is caused by LOW secretion of adrenal hormones β€” glucocorticoids, mineralocorticoids, and androgens. The body lacks the hormones it needs to maintain blood pressure, blood sugar, and electrolyte balance. Addisonian crisis is a life-threatening emergency.
S
Sugar and Sodium low β€” hypoglycemia, hyponatremia
T
Tired and muscle weakness β€” profound fatigue
E
Electrolyte imbalance β€” high K+, high Ca2+
R
Reproductive changes β€” decreased libido
O
lOw blood pressure β€” orthostatic hypotension
I
Increased skin pigmentation β€” bronze appearance
D
Diarrhea, nausea, Depression
πŸ“– Full Lesson β†’
Multiple Sclerosis
DEMYELINATION
Diplopia Β· Eye pain Β· Motor weakness Β· nYstagmus Β· Elevated temp Β· Lhermitte's Β· Neuropathic pain Β· Ataxia Β· Talking slurred Β· Impotence Β· Overactive bladder Β· Numbness
Common symptoms of multiple sclerosis
Multiple sclerosis causes demyelination in the central nervous system β€” destroying the protective myelin sheath around nerves. DEMYELINATION spells out its own symptoms, making this one of the most elegant mnemonics in nursing. Symptoms worsen with heat (Uhthoff's phenomenon).
D
Diplopia β€” double vision
E
Eye movement painful β€” optic neuritis
M
Motor β€” weakness and spasticity
Y
nYstagmus β€” involuntary eye movement
E
Elevated temperature worsens symptoms
L
Lhermitte's β€” electric shock with neck flexion
I
Impotence β€” sexual dysfunction
N
Neuropathic pain
A
Ataxia β€” balance and coordination problems
T
Talking slurred β€” dysarthria
I
Overactive bladder β€” urgency, incontinence
O
Numbness and tingling
N
Numbness β€” paresthesias throughout body
πŸ“– Full Lesson β†’
Anion Gap Metabolic Acidosis
GOLDMARK
Glycols Β· Oxoproline Β· L-lactate Β· D-lactate Β· Methanol Β· Aspirin Β· Renal failure Β· Ketoacidosis
Causes of anion gap metabolic acidosis
GOLDMARK helps you remember the causes of anion gap metabolic acidosis β€” when the kidneys are not removing enough acid from the body. Essential for med-surg and critical care nursing.
G
Glycols β€” ethylene glycol and propylene glycol poisoning
O
Oxoproline β€” accumulates with chronic acetaminophen use
L
L-lactate β€” lactic acidosis from shock or hypoxia
D
D-lactate β€” from short bowel syndrome
M
Methanol β€” toxic alcohol ingestion
A
Aspirin β€” salicylate toxicity
R
Renal failure or uremia β€” kidneys can't excrete acid
K
Ketoacidosis β€” DKA, alcoholic, starvation
πŸ“– Full Lesson β†’
Cholinergic Crisis
SLUDGE
Salivation Β· Lacrimation Β· Urination Β· Defecation Β· Gastric upset Β· Emesis
Signs and symptoms of cholinergic crisis
SLUDGE helps identify a cholinergic crisis β€” when the body fails to break down acetylcholine properly. This can occur with organophosphate poisoning or certain medications. Antidote is atropine.
S
Salivation β€” excessive drooling
L
Lacrimation β€” excessive tearing
U
Urination β€” urinary incontinence
D
Defecation β€” diarrhea
G
Gastric upset β€” nausea, cramping
E
Emesis β€” vomiting
πŸ“– Full Lesson β†’
Epiglottitis
AIR RAID
Airway closed Β· Increased pulse Β· Restlessness Β· Retractions Β· Anxiety Β· Inspiratory stridor Β· Drooling
Recognize epiglottitis β€” a life-threatening airway emergency
AIR RAID helps identify epiglottitis β€” inflammation of the epiglottis blocking the airway. This is a medical emergency requiring immediate intervention. Do NOT attempt to visualize the throat β€” it can cause complete airway obstruction.
A
Airway closed β€” obstructed by swollen epiglottis
I
Increased pulse β€” tachycardia from distress
R
Restlessness β€” agitation from hypoxia
R
Retractions β€” intercostal muscles pulling inward
A
Anxiety β€” air hunger and fear
I
Inspiratory stridor β€” turbulent airflow through larynx
D
Drooling β€” unable to swallow secretions
πŸ“– Full Lesson β†’
Family Medical History
BALD CHASM
Blood pressure Β· Arthritis Β· Lung diseases Β· Diabetes Β· Cancers Β· Heart diseases Β· Alcoholism Β· Stroke Β· Mental health disorders
Assess family history for genetically transmitted conditions
BALD CHASM helps you systematically assess a patient's family medical history for hereditary conditions. A thorough family history can reveal risk factors that shape your entire care plan.
B
Blood pressure β€” hypertension history
A
Arthritis β€” rheumatoid or osteoarthritis
L
Lung diseases β€” COPD, asthma, TB
D
Diabetes β€” Type 1 or Type 2
C
Cancers β€” any type in family history
H
Heart diseases β€” MI, CHF, arrhythmias
A
Alcoholism β€” substance use disorders
S
Stroke β€” CVA history
M
Mental health disorders β€” depression, schizophrenia
πŸ“– Full Lesson β†’
Body Systems
MR. DICE RUNS
Muscular Β· Respiratory Β· Digestive Β· Integumentary Β· Circulatory Β· Endocrine Β· Reproductive Β· Urinary Β· Nervous Β· Skeletal
Remember all 10 body systems for exams
MR. DICE RUNS is a fun and memorable way to recall all 10 major body systems. Knowing these systems is foundational for med-surg nursing and essential for NCLEX preparation.
M
Muscular
R
Respiratory
D
Digestive
I
Integumentary β€” skin, hair, nails
C
Circulatory
E
Endocrine
R
Reproductive
U
Urinary
N
Nervous
S
Skeletal
πŸ“– Full Lesson β†’
MI β€” MONA
MI treatment: MONA β€” Morphine, Oxygen, Nitrates, Aspirin. 12-lead ECG within 10 min. Troponin is gold standard.
Myocardial Infarction
Recognizing and responding to MI β€” time is muscle, every minute counts
Classic MI symptoms: crushing chest pain (may radiate to jaw, left arm, back), diaphoresis, nausea, shortness of breath. Women/diabetics: atypical β€” jaw pain, fatigue, nausea only. MONA: Morphine (reduces preload/pain β€” now questioned in STEMI), Oxygen (if SpO2 <90%), Nitrates (vasodilation β€” hold if systolic <90 or recent sildenafil use), Aspirin (antiplatelet β€” 325 mg chewed). ECG: within 10 minutes of arrival. ST elevation = STEMI β€” needs cath lab within 90 min. Troponin: gold standard biomarker, rises 3–4 hr after MI. Give nothing by mouth (NPO) β€” may need procedure.
M
Morphine β€” pain/anxiety
O
Oxygen β€” if SpO2 <90%
N
Nitrates β€” vasodilate, hold if BP low
A
Aspirin 325 mg chewed
πŸ“– Full Lesson β†’
Stroke β€” FAST
Stroke: FAST β€” Face drooping, Arm weakness, Speech difficulty, Time to call 911. tPA within 3–4.5 hours of onset.
Stroke Recognition and Care
Time-critical emergency β€” recognizing stroke and the nursing response
FAST: Face drooping (ask to smile β€” asymmetry), Arm weakness (raise both arms β€” one drifts down), Speech difficulty (slurred or unable to speak), Time β€” call 911 immediately. Two types: Ischemic (87% β€” clot) and Hemorrhagic (13% β€” bleed). Treatment: Ischemic β†’ tPA (alteplase) if within 3–4.5 hours of LAST KNOWN WELL, no hemorrhage on CT. Hemorrhagic β†’ no tPA, manage BP, possible surgery. Nursing: NIH Stroke Scale assessment, position HOB 30Β°, NPO until swallow evaluation, falls precautions, BP management (allow permissive hypertension in ischemic unless giving tPA). Time is brain β€” 1.9 million neurons lost per minute.
F
Face drooping
A
Arm weakness
S
Speech difficulty
T
Time β€” call 911 now
tPA window
3–4.5 hours from last known well
πŸ“– Full Lesson β†’
Increased Intracranial Pressure
ICP signs: Cushing's Triad β€” Bradycardia, Hypertension (widening pulse pressure), Irregular respirations. LATE sign = impending herniation.
Increased ICP
Cushing's Triad is a medical emergency β€” the nurse must recognize it and act immediately
Normal ICP: 5–15 mmHg. Early ICP signs: headache (worse with straining), nausea/vomiting (projectile), altered LOC, pupil changes (unequal, sluggish). Late sign β€” Cushing's Triad (EMERGENCY): Bradycardia + Hypertension (widening pulse pressure) + Irregular/slow respirations β†’ impending brainstem herniation. Nursing: HOB 30Β°, head midline (no neck rotation β€” impairs venous drainage), avoid clustering care, dim lights/quiet environment, avoid Valsalva (no straining), monitor pupil response. Do NOT: suction vigorously, hip flexion >90Β°, prone positioning.
Early
Headache, N/V, altered LOC
Cushing's Triad
Bradycardia + HTN + irregular RR
Position
HOB 30Β°, head midline
Avoid
Straining, Valsalva, clustering care
πŸ“– Full Lesson β†’
Respiratory β€” COPD vs Asthma
COPD: chronic, progressive, barrel chest, pursed-lip breathing. Asthma: episodic, reversible, wheezing, triggered. O2 cautiously in COPD.
COPD vs Asthma
Two obstructive lung diseases with important differences β€” NCLEX loves the oxygen question in COPD
COPD (emphysema + chronic bronchitis): irreversible airway obstruction. Emphysema: barrel chest, pursed-lip breathing, decreased breath sounds, 'pink puffer' (fights to breathe). Chronic bronchitis: productive cough >3 months/2 years, 'blue bloater.' O2 in COPD: hypoxic drive β€” give O2 2–3 L/NC, target SpO2 88–92% (not 95–100%). High O2 may suppress respiratory drive. Asthma: reversible bronchospasm, triggered (allergens, exercise, cold). Wheezing on expiration. Peak flow meter: green >80%, yellow 50–80%, red <50%. Rescue inhaler (albuterol) before preventive (corticosteroid inhaler).
COPD O2
2–3 L, target SpO2 88–92%
Emphysema
Barrel chest, pursed lips, pink puffer
Chronic Bronchitis
Productive cough, blue bloater
Asthma
Reversible, wheezing, rescue before preventive
πŸ“– Full Lesson β†’
Pneumonia
Pneumonia assessment: fever, productive cough, crackles, decreased breath sounds. Position: semi-Fowler's. Encourage fluids and deep breathing.
Pneumonia Nursing
The most common hospital-acquired infection β€” assessment, positioning, and prevention
Signs: fever and chills, productive cough (yellow/green/rust-colored sputum), pleuritic chest pain (worse with breathing), crackles and decreased breath sounds in affected lobe, tachypnea, hypoxia. Community-acquired (CAP): S. pneumoniae most common. Hospital-acquired (HAP): gram-negative organisms, MRSA. Nursing care: semi-Fowler's position (HOB 30–45Β°), encourage fluids (2–3 L/day unless restricted β€” thins secretions), deep breathing and coughing exercises, incentive spirometer, turn every 2 hours, ambulate early. Prevention: pneumococcal vaccine, hand hygiene, oral care in ventilated patients (VAP bundle).
Assessment findings
Fever, productive cough, crackles/rhonchi, decreased breath sounds
Community vs hospital
CAP most common: Strep pneumoniae. HAP: Pseudomonas and MRSA
Nursing interventions
HOB up 30–45Β°, encourage deep breathing, hydration, incentive spirometry
Antibiotics
Start within 4 hours of diagnosis; do NOT delay for culture results in severe cases
πŸ“– Full Lesson β†’
Diabetes β€” Hypo vs Hyperglycemia
Hypoglycemia (<70): Cold and Clammy = give candy. Hyperglycemia (>180): Hot and Dry = sugar high.
Hypoglycemia vs Hyperglycemia
The quick way to distinguish and treat two dangerous blood sugar extremes
Hypoglycemia (<70 mg/dL): Cold and Clammy β€” diaphoresis, tremors, tachycardia, confusion, seizure. Cause: too much insulin, missed meal, excess exercise. Treatment: 15-15 rule β€” 15g fast carbs (4 oz juice, glucose tablets), recheck in 15 min. If unconscious: IV dextrose (D50) or glucagon IM. Hyperglycemia (>180–250): Hot and Dry β€” polyuria (3 Ps: Polyuria, Polydipsia, Polyphagia), fruity breath (DKA), Kussmaul respirations (deep, rapid β€” blowing off CO2 in DKA). DKA (Type 1): ketones, pH <7.3. HHS (Type 2): extreme hyperglycemia, no ketones, elderly. Treatment: insulin drip, IV fluids, K+ replacement.
Hypo <70
Cold/clammy, diaphoresis, tremor, confusion
Hypo Tx
15g carbs β†’ recheck 15 min, or IV D50
Hyper
Hot/dry, 3 Ps, fruity breath
DKA
Ketones, Kussmaul breathing, pH <7.3
πŸ“– Full Lesson β†’
Renal Failure β€” AEIOU
AEIOU β€” Acute kidney injury complications: Acidosis, Electrolyte imbalances (K+↑), Intoxication (uremia), Overload (fluid), Uremia.
Acute Kidney Injury
Recognizing and managing AKI β€” the NCLEX expects nurses to monitor and intervene
AKI: rapid decline in kidney function over hours to days. Stages (RIFLE/KDIGO): Risk, Injury, Failure, Loss, ESKD. Oliguric phase: urine output <0.5 mL/kg/hr, BUN and creatinine rise, K+ rises (hyperkalemia β€” most dangerous). AEIOU complications: Acidosis (metabolic), Electrolyte imbalance (hyperkalemia β†’ EKG changes, peaked T-waves β†’ cardiac arrest), Intoxication (uremia β€” confusion, asterixis), Overload (fluid), Uremia (N/V, pericarditis, pruritus). Nursing: strict I&O, daily weights, low K+ diet, BP monitoring, dialysis access care. Fluid challenge: 500 mL NS bolus if pre-renal cause.
A
Acidosis β€” metabolic
E
Electrolytes β€” hyperkalemia, peaked T waves
I
Intoxication β€” uremia, confusion
O
Overload β€” fluid
U
Uremia β€” N/V, pericarditis, pruritus
πŸ“– Full Lesson β†’
Postoperative Care
Post-op ABCDE: Airway, Breathing, Circulation, Drugs (anesthesia), Everything else (pain, N/V, wound).
Postoperative Nursing
Systematic assessment immediately after surgery β€” and the complications to watch for
Immediate post-op (PACU): Airway β€” maintain, suction if needed. Breathing β€” respiratory rate, SpO2, breath sounds. Circulation β€” BP, HR, bleeding at surgical site. Drugs β€” anesthesia reversal, pain management. Temperature β€” hypothermia common (warm blankets, forced air). Early complications: respiratory depression (opioids β€” give Narcan), airway obstruction (tongue, secretions), hemorrhage (increasing HR, decreasing BP), emergence delirium. Late complications: atelectasis (encourage deep breathing, IS), DVT (SCDs, early ambulation, anticoagulants), wound infection (3–5 days post-op fever), paralytic ileus (listen for bowel sounds).
A β€” Airway
Ensure patent airway; have suction ready; semi-conscious patients at risk
B β€” Breathing
Monitor RR, SpO2, symmetrical chest rise; deep breathing every hour
C β€” Circulation
VS every 15 min x4, then 30 min x4; monitor dressing for bleeding
D β€” Drugs
Assess anesthesia reversal; pain management; antiemetics for nausea
E β€” Environment
Warm blankets β€” hypothermia common post-op; minimize stimulation
πŸ“– Full Lesson β†’
Fluid and Electrolytes β€” Big 5
Hypokalemia: U waves, weak muscles. Hyperkalemia: peaked T waves. Hyponatremia: confusion, seizures. Hypernatremia: thirst, dry mucosa.
Electrolyte Imbalances
The five electrolytes NCLEX tests most β€” know the critical values and EKG changes
Potassium (normal 3.5–5.0): Hypo (<3.5): muscle weakness, cramps, U waves on EKG, constipation. Causes: diuretics, vomiting, NG suction. Replace slowly (never IV push β€” fatal). Hyper (>5.5): peaked T waves, wide QRS, muscle weakness, cardiac arrest. Treat: calcium gluconate (protect heart), insulin+dextrose (shift K+ into cells), Kayexalate. Sodium (normal 135–145): Hypo (<135): headache, confusion, seizures β€” restrict fluids, hypertonic saline (slowly or central herniation). Hyper (>145): thirst, dry mucosa, restlessness, seizures β€” free water replacement. Calcium: Hypo β€” Trousseau's and Chvostek's signs, tetany. Magnesium: Hypo β€” cardiac dysrhythmias.
Hypokalemia
U waves, muscle weak β€” diuretics cause
Hyperkalemia
Peaked T waves β€” cardiac emergency
Hyponatremia
Confusion, seizures β€” fluid restrict
Hypernatremia
Thirst, dry β€” give free water slowly
πŸ“– Full Lesson β†’
Wound Care and Pressure Injuries
Pressure injury stages: I (redness), II (partial thickness), III (full thickness), IV (bone/tendon visible). Turn every 2 hours.
Pressure Injuries
Staging wounds and preventing pressure injuries β€” prevention is always better than treatment
Stage I: intact skin, non-blanchable redness. Intervention: relieve pressure, moisturize. Stage II: partial thickness skin loss β€” shallow open ulcer or blister. Stage III: full thickness skin loss, subcutaneous tissue visible, no bone/tendon. Stage IV: full thickness, bone/tendon/muscle exposed. Unstageable: covered by eschar β€” cannot stage until debrided. Deep tissue injury (DTI): purple/maroon discoloration, intact skin. Prevention: turn every 2 hours, pressure-relieving mattress, keep dry (moisture = skin breakdown), adequate nutrition (protein + vitamin C + zinc), assess Braden scale. Never massage over bony prominences β€” increases breakdown.
Stage I
Non-blanchable redness β€” intact skin
Stage II
Blister or shallow ulcer
Stage III
Full thickness β€” no bone visible
Stage IV
Bone/tendon visible
Prevention
Turn q2h, Braden scale, nutrition
πŸ“– Full Lesson β†’
ABG Interpretation
ABGs: pH 7.35–7.45, PaCO2 35–45, HCO3 22–26. ROME: Respiratory Opposite, Metabolic Equal.
ABG Interpretation
The step-by-step method for reading arterial blood gases β€” ROME makes it systematic
Normal values: pH 7.35–7.45, PaCO2 35–45 mmHg (respiratory), HCO3 22–26 mEq/L (metabolic). Step 1: pH β€” acidosis (<7.35) or alkalosis (>7.45)? Step 2: PaCO2 β€” if it matches pH direction (opposite), it's respiratory. Step 3: HCO3 β€” if it matches pH direction (same), it's metabolic. ROME: Respiratory Opposite (pH up, CO2 down = alkalosis), Metabolic Equal (pH up, HCO3 up = alkalosis). Compensation: the system NOT causing the problem tries to correct pH. Respiratory acidosis (hypoventilation, COPD): pH↓, CO2↑. Metabolic acidosis (DKA, renal failure): pH↓, HCO3↓. Metabolic alkalosis (vomiting, NG suction): pH↑, HCO3↑.
pH <7.35
Acidosis
pH >7.45
Alkalosis
CO2 matches pH?
Respiratory cause
HCO3 matches pH?
Metabolic cause
ROME
Respiratory Opposite, Metabolic Equal
πŸ“– Full Lesson β†’
🩸 Clot
DVT = Deep Vein Thrombosis β†’ PE = Pulmonary Embolism β€” one feeds the other
DVT AND PULMONARY EMBOLISM
DVT and PE β€” recognition, prevention, and the nursing response
DVT risk (Virchow's Triad): Stasis (immobility, bedrest, long travel), Vessel damage (trauma, surgery, IV access), Hypercoagulability (pregnancy, cancer, clotting disorders, OCP). DVT signs: unilateral leg swelling, warmth, redness, pain β€” Homan's sign (calf pain with dorsiflexion) unreliable but still tested. PE signs: sudden dyspnea, pleuritic chest pain, tachycardia, hypoxia, hemoptysis, anxiety β€” can cause sudden death. PE triad: dyspnea + chest pain + hemoptysis. Prevention: early ambulation, SCDs (sequential compression devices), Ted hose, hydration, anticoagulation. Treatment: heparin β†’ warfarin or LMWH β†’ DOAC. Nursing: never massage a suspected DVT (embolization risk), elevate extremity, warm compresses, monitor anticoagulation levels, bleeding precautions. NCLEX: sudden onset dyspnea post-op = PE until proven otherwise.
DVT
Unilateral leg swelling, warmth, pain, redness
PE
Sudden dyspnea, chest pain, tachycardia, hypoxia
Never do
Massage DVT β€” dislodges clot β†’ PE
Post-op rule
Sudden dyspnea = PE until proven otherwise
πŸ“– Full Lesson β†’
πŸ”₯ Thyroid Crisis
Storm = everything UP. Coma = everything DOWN. Both are life-threatening.
THYROID STORM vs MYXEDEMA COMA
Thyroid storm vs myxedema coma β€” the two thyroid emergencies NCLEX tests
Thyroid Storm (thyrotoxic crisis): severe hyperthyroidism β€” triggered by stress, infection, surgery. Signs: hyperthermia (high fever), tachycardia (often A-fib), HTN, agitation, diaphoresis, tremor, diarrhea, vomiting β€” can progress to heart failure and death. Treatment: propylthiouracil (PTU) or methimazole (block new hormone), propranolol (control HR), glucocorticoids, cooling measures, treat precipitating cause. Myxedema Coma: severe hypothyroidism β€” triggered by cold, infection, sedatives. Signs: hypothermia, bradycardia, hypotension, hypoventilation, hyponatremia, altered LOC, myxedema (non-pitting facial/extremity edema). Treatment: IV levothyroxine, warming blankets (slowly β€” rapid rewarming causes vasodilation), airway management, corticosteroids. NCLEX: both are emergencies. Storm = everything elevated. Coma = everything depressed.
Storm triggers
Infection, surgery, trauma, abrupt medication discontinuation
Storm = everything UP
HR up, temp up, BP up, metabolic rate up, agitation, tremors
Myxedema coma = everything DOWN
HR down, temp down, BP down, lethargy, hypoventilation
Storm treatment
PTU, beta-blocker, corticosteroids, cooling, iodine
Coma treatment
IV levothyroxine, warming, IV fluids, steroids, possible intubation
πŸ“– Full Lesson β†’
πŸ”₯ Burns
Rule of Nines β€” head 9%, each arm 9%, each leg 18%, front torso 18%, back 18%, perineum 1%
BURN ASSESSMENT AND FLUID RESUSCITATION
Burns β€” classification, Rule of Nines, and the Parkland formula for fluid resuscitation
Burn depth: Superficial (1st degree) β€” epidermis only, red, painful, no blisters (sunburn). Partial thickness (2nd degree) β€” epidermis + dermis, blisters, moist, very painful β€” most painful burn. Full thickness (3rd degree) β€” all layers, leathery, dry, painless (nerve destruction), requires grafting. 4th degree β€” bone/muscle involvement. Rule of Nines (adults): Head/neck = 9%, Each arm = 9%, Chest = 18%, Back = 18%, Each leg = 18%, Perineum = 1%. Fluid resuscitation (Parkland formula): 4 mL x kg x %TBSA burned. Give 1/2 in first 8 hours from time of injury (NOT from hospital arrival), remaining 1/2 over next 16 hours. Use Lactated Ringer's. Monitor urine output (goal 0.5–1 mL/kg/hr adults). Airway is priority β€” inhalation injury kills. Circumferential burns may need escharotomy.
Head/neck
9%
Each arm
9% (18% total)
Anterior/posterior torso
18% each (36% total)
Each leg
18% (36% total)
Perineum
1%
πŸ“– Full Lesson β†’
🩸 DIC
DIC = Death Is Coming β€” bleed everywhere AND clot everywhere simultaneously
DISSEMINATED INTRAVASCULAR COAGULATION
DIC β€” the paradoxical clotting disorder where patients bleed and clot at the same time
DIC is not a disease β€” it's a complication of: sepsis (most common), obstetric emergencies (abruption, amniotic fluid embolism, PPH), trauma, massive transfusion, cancer. Pathophysiology: massive coagulation cascade activation β†’ consumes all clotting factors and platelets β†’ simultaneous microvascular clotting AND uncontrolled bleeding. Signs: bleeding from all sites (IV sites, gums, nose, petechiae, purpura, hematuria, GI bleed) AND organ ischemia from microthrombi (necrosis, renal failure). Labs: PT↑, PTT↑, platelets↓, fibrinogen↓, D-dimer↑ (markedly), schistocytes on smear. Treatment: treat underlying cause, replace clotting factors (FFP, cryoprecipitate, platelets), RBC transfusion, heparin controversial. Nursing: assess all body systems for bleeding, pad side rails, gentle handling, no IM injections, pressure on puncture sites.
Pathophysiology
Widespread clotting consumes clotting factors causing paradoxical bleeding
Triggers
Sepsis, trauma, obstetric emergencies, transfusion reaction
Labs
PT up, PTT up, fibrinogen DOWN, D-dimer UP, platelets DOWN
Clinical signs
Oozing from IV sites, petechiae, ecchymosis, hematuria, organ failure
Treatment
Treat underlying cause first; FFP, cryoprecipitate, platelets as ordered
πŸ“– Full Lesson β†’
😨 Shock
All shock = low perfusion. Cause differs: volume, pump, distribution, or obstruction.
TYPES OF SHOCK AND NURSING PRIORITIES
The four types of shock β€” distinguishing features and priority nursing interventions
All shock = inadequate tissue perfusion β†’ cellular hypoxia β†’ organ failure. Hypovolemic: low volume (hemorrhage, burns, dehydration) β€” cold/clammy, tachycardia, low BP. Treat: fluids, blood, control bleeding. Cardiogenic: pump failure (MI, heart failure) β€” cold/clammy, elevated JVD, pulmonary edema. Treat: inotropes, diuretics (not fluids!). Distributive (septic, neurogenic, anaphylactic): vasodilation β†’ maldistribution β€” warm/flushed early. Septic: fever, tachycardia, hypotension, altered LOC β€” sepsis bundle (blood cultures, broad antibiotics, fluids, vasopressors). Neurogenic (spinal injury): hypotension + bradycardia (no tachycardia β€” lost sympathetic tone) + warm/dry skin. Anaphylactic: epinephrine IM first, airway, diphenhydramine, steroids. Obstructive: mechanical obstruction (PE, tension pneumo, cardiac tamponade) β€” treat cause. Universal shock nursing: large-bore IV x2, fluid challenge (except cardiogenic), O2, VS Q15min, urine output, elevate legs (except cardiogenic/neuro).
Hypovolemic
Cold/clammy, tachy β€” fluids and blood
Cardiogenic
Cold/clammy + JVD/crackles β€” no fluids
Septic
Warm early, cold late β€” cultures then abx
Neurogenic
Hypotension + bradycardia β€” unique finding
Anaphylactic
Epi IM first β€” airway priority
πŸ“– Full Lesson β†’
🧠 Neuro
FAST + BE-FAST β€” Balance, Eyes, Face, Arm, Speech, Time β€” stroke recognition
INCREASED INTRACRANIAL PRESSURE (ICP)
Increased ICP β€” early vs late signs and the nursing interventions that matter
Normal ICP: 5–15 mmHg. Causes: head trauma, hemorrhage, tumor, hydrocephalus, meningitis, stroke. Early signs: headache (worse in morning, with Valsalva), nausea/vomiting (projectile, without nausea), changes in LOC (earliest sign β€” restlessness, confusion), pupil changes (sluggish), blurred/double vision. Late signs (herniation imminent): Cushing's Triad = hypertension + bradycardia + irregular respirations. Decorticate posturing (arms flexed = cortical damage), Decerebrate posturing (arms extended = brainstem damage β€” worse). Nursing interventions: HOB 30–45Β° (promotes venous drainage), neck in neutral alignment, minimize stimulation, avoid Valsalva (no straining, coughing β€” stool softeners), cluster care, avoid hyperthermia, monitor neuro status Q1h, avoid hypotonic fluids (worsens cerebral edema). Medications: mannitol (osmotic diuretic), hypertonic saline, corticosteroids (tumor only).
Early
LOC change (first!), headache, N/V, pupil sluggish
Late
Cushing's triad, posturing, coma
HOB
30–45Β° β€” neutral neck alignment
Avoid
Valsalva, hyperthermia, clustering stimuli
πŸ“– Full Lesson β†’
πŸ₯ Sepsis
SIRS + suspected infection = Sepsis. Time is tissue β€” the Hour-1 Bundle saves lives.
SEPSIS RECOGNITION AND THE HOUR-1 BUNDLE
Sepsis β€” early recognition, qSOFA, and the nursing actions in the first hour
Sepsis = life-threatening organ dysfunction from dysregulated response to infection. qSOFA screening (2 of 3 = high risk): Altered mental status, Respiratory rate β‰₯22, Systolic BP ≀100. Septic shock = sepsis + vasopressors needed to maintain MAP β‰₯65 + lactate >2 despite fluids. Hour-1 Bundle (Surviving Sepsis Campaign): Measure lactate (>2 = tissue hypoperfusion), Obtain blood cultures x2 before antibiotics, Administer broad-spectrum antibiotics, Begin 30 mL/kg crystalloid for hypotension or lactate β‰₯4, Apply vasopressors (norepinephrine first-line) for MAP <65. Nursing: recognize early (subtle LOC change, tachycardia, fever or hypothermia, tachypnea), act fast, large-bore IV access, urine output monitoring (goal >0.5 mL/kg/hr), repeat lactate at 2 hours if initial >2. Document time of all bundle elements.
Sepsis definition
SIRS + suspected or confirmed infection source
Septic shock
Sepsis + hypotension despite fluid resuscitation + lactate above 2
Hour-1 bundle
Blood cultures x2, lactate, broad-spectrum antibiotics, 30 mL/kg IV fluid bolus
Nursing priority
TIME IS CRITICAL β€” each hour of delay in antibiotics increases mortality 7%
πŸ“– Full Lesson β†’
🫁 Pneumothorax
Tension pneumo = tracheal deviation AWAY from affected side β€” needle decompression NOW
PNEUMOTHORAX AND CHEST TUBE CARE
Pneumothorax types and chest tube nursing β€” the complications that kill and how to prevent them
Pneumothorax = air in pleural space β†’ lung collapse. Simple: spontaneous (tall thin young males) or traumatic. Tension: air enters but cannot escape β†’ mediastinal shift β†’ compresses heart and great vessels β†’ obstructive shock. EMERGENCY β€” tracheal deviation AWAY from affected side, absent breath sounds, JVD, hypotension, tachycardia. Treatment: needle decompression (2nd ICS, MCL) β†’ chest tube. Chest tube nursing: keep drainage system below chest level, keep tubing free of kinks, tidaling normal (fluid rises with inspiration, falls with expiration β€” means patent), bubbling in water seal = air leak (continuous = bad, intermittent normal during coughing). Clamping = dangerous β€” do not clamp without order. If tube dislodges: cover with petroleum gauze taped on 3 sides (flutter valve). Monitor drainage β€” notify if >100 mL/hr (hemorrhage).
Simple pneumo
Air in pleural space; decreased breath sounds; trachea midline
Tension pneumo
Trachea deviates AWAY from affected side; JVD; hemodynamic instability
Emergency action
Tension pneumo = needle decompression 2nd ICS midclavicular line IMMEDIATELY
Chest tube care
Water seal chamber bubbles with exhalation; tidaling is normal; no dependent loops
Never clamp
Do NOT clamp chest tube β€” risk of tension pneumo
πŸ“– Full Lesson β†’
Stroke Recognition
FAST
Face drooping Β· Arm weakness Β· Speech difficulty Β· Time to call 911
Every Second Counts β€” Teach Every Patient This
Stroke is a "Time = Brain" emergency β€” 1.9 million neurons die every minute without treatment. The thrombolytic window for tPA is 3–4.5 hours from symptom onset. FAST helps bystanders AND nurses recognize stroke fast. In hospital, use the NIH Stroke Scale. New onset facial droop + arm drift + slurred speech = stroke until proven otherwise β€” call a rapid response immediately.
F
Face β€” ask patient to smile; drooping on one side?
A
Arms β€” raise both; does one drift downward?
S
Speech β€” slurred, strange, unable to speak or repeat?
T
Time β€” note symptom onset; call rapid response/911
πŸ“– Full Lesson β†’
DVT Prevention
Virchow's Triad
Stasis Β· Hypercoagulability Β· Vessel wall injury
Why DVTs Form β€” And How to Stop Them
Virchow's Triad explains every DVT. Post-op patients hit all three: immobility (stasis), trauma (vessel injury), surgical stress response (hypercoagulability). Prevention bundle: early ambulation, sequential compression devices (SCDs), anticoagulants. Never massage a suspected DVT β€” risk of pulmonary embolism. Homan's sign is unreliable β€” don't rely on it.
1
Venous stasis β€” immobility, prolonged bed rest, long travel
2
Hypercoagulability β€” post-op, cancer, pregnancy, oral contraceptives
3
Vessel wall injury β€” trauma, surgery, IV line placement
πŸ“– Full Lesson β†’
Delirium vs Dementia
ACUTE vs CHRONIC
Delirium = Acute Β· Reversible Β· Fluctuating β€” Dementia = Chronic Β· Progressive Β· Stable daily
The Most Commonly Confused Diagnosis Pair
The key differentiator: ONSET. Delirium = hours to days, fluctuates, has a cause (infection, meds, pain, hypoxia). Dementia = months to years, steady decline. A delirious patient with known dementia is common β€” always look for the NEW reversible cause. Use I WATCH DEATH to find it. Treat the cause, not just the symptoms.
D
Delirium β€” sudden onset, reversible, fluctuates hour to hour
D
Dementia β€” gradual onset, irreversible, slowly progressive
!
Both β€” delirium ON TOP of dementia is extremely common
πŸ“– Full Lesson β†’
🎓 Common Exam Questions
Q: What are the differences between DKA and HHS?
A: DKA (Type 1 typically): glucose above 250, pH below 7.3, ketones positive, anion gap elevated. HHS (Type 2 typically): glucose above 600, pH normal, no ketones, extreme hyperosmolarity. Both: IV fluids first, then insulin drip, then electrolyte replacement β€” watch potassium closely.
Q: What are the nursing priorities for a patient with acute MI?
A: MONA: Morphine, Oxygen (if SpO2 below 90%), Nitrates (hold if SBP below 90 or right-sided MI), Aspirin (325mg chewed). Priority: 12-lead ECG within 10 minutes, troponin, IV access, continuous cardiac monitoring, prepare for PCI β€” door-to-balloon goal under 90 minutes.
Q: How do you manage a patient in heart failure?
A: Daily weight (report above 2 lbs/day gain), fluid restriction (1.5-2L/day), low sodium diet, diuretics (monitor potassium), ACE inhibitors or ARBs, beta-blockers (NOT in acute decompensation). Left-sided HF: dyspnea, crackles β€” high Fowler's. Right-sided HF: JVD, peripheral edema.
Q: What is the nurse's role managing a patient with a chest tube?
A: Normal: gentle bubbling with exhalation, tidaling. Concerning: continuous bubbling at rest = air leak; no tidaling = kinked tube. Never clamp without specific orders. Report drainage above 100 mL/hr of blood. Keep drainage system below chest at all times.
Q: What are the signs of increased ICP and nursing priorities?
A: Early: headache, nausea, vomiting, LOC changes, pupil changes. Cushing's triad (emergency): hypertension + bradycardia + irregular respirations. Nursing: HOB at 30-45Β°, head midline, avoid Valsalva, maintain SpO2 above 94%, avoid hyperthermia, continuous neuro checks.