Four stages every OB nurse must know β with the key assessments for each
First stage: latent (0β6 cm, irregular contractions), active (6β10 cm, stronger/closer contractions), transition (8β10 cm, most intense). Assess: cervical dilation, effacement, station, fetal heart rate, contractions. Second stage: complete dilation to birth. Push with contractions (closed glottis). Monitor fetal heart rate. Third stage: placenta delivery β within 30 minutes. Signs of separation: gush of blood, lengthening of cord, uterine fundus rises and becomes firm. Fourth stage: first 1β4 hours after delivery. Assess: fundus (firm, midline, at umbilicus), lochia, perineum, BP, HR. Most common time for postpartum hemorrhage.
1st stage
0β10 cm dilation
2nd stage
Pushing β delivery of baby
3rd stage
Placenta delivery β <30 min
4th stage
First 1β4 hrs β hemorrhage risk
Fetal Heart Rate Patterns
FHR decelerations: Early (head compression β normal), Variable (cord compression β change position), Late (uteroplacental insufficiency β EMERGENCY).
Fetal Heart Rate Decelerations
Three deceleration patterns β one is normal, one needs repositioning, one is an emergency
Early decelerations: mirror contractions (start and end together), caused by head compression, normal β no intervention needed. Variable decelerations: abrupt drop, variable timing, caused by cord compression. Intervention: change maternal position (left lateral, knee-chest), O2, stop oxytocin, fluid bolus β may need amnioinfusion. Late decelerations: begin AFTER peak of contraction, caused by uteroplacental insufficiency (placenta not delivering enough O2 to fetus). EMERGENCY β notify provider immediately. Interventions: left lateral position, O2 10 L nonrebreather, stop oxytocin, IV fluid bolus, prepare for delivery. Persistent late decels = C-section.
Early
Head compression β mirror contraction, NORMAL
Variable
Cord compression β reposition, O2
Late
Uteroplacental insufficiency β EMERGENCY
Postpartum Hemorrhage
PPH: blood loss >500 mL vaginal, >1000 mL C-section. 4 Ts: Tone (uterine atony #1), Trauma, Tissue, Thrombin.
Postpartum Hemorrhage
The leading cause of maternal mortality β recognizing and responding to PPH
Most common cause: uterine atony (boggy uterus = not contracting). 4 Ts: Tone (70β80% β uterine atony), Trauma (lacerations), Tissue (retained placenta), Thrombin (coagulopathy). Assessment: fundus (boggy? midline? above umbilicus?), lochia (saturating pad in <1 hour = abnormal), vital signs (tachycardia first sign, then hypotension). Management of atony: massage fundus (never pummel), bimanual compression, oxytocin (Pitocin), methergine (not in HTN), carboprost (not in asthma), Bakri balloon, blood products. Monitor: I&O, H&H, coagulation studies.
Tone
Uterine atony β most common cause
Trauma
Lacerations
Tissue
Retained placenta
Thrombin
Coagulopathy
Preeclampsia
Preeclampsia: BP β₯140/90 after 20 weeks + proteinuria. Severe: BP β₯160/110, HELLP syndrome, seizure (eclampsia) = EMERGENCY.
Preeclampsia and Eclampsia
The hypertensive disorder of pregnancy that can progress to life-threatening eclampsia
Preeclampsia: onset >20 weeks gestation. Criteria: BP β₯140/90 on two occasions 4 hrs apart, plus proteinuria. Severe features: BP β₯160/110, platelets <100,000, creatinine >1.1, LFTs 2Γ normal, pulmonary edema, severe headache, visual disturbances, RUQ pain. HELLP syndrome: Hemolysis, Elevated Liver enzymes, Low Platelets. Nursing: quiet, dim room (reduce stimuli), magnesium sulfate (seizure prophylaxis AND treatment β monitor for toxicity: absent DTRs, RR <12, urine <25 mL/hr), antidote = calcium gluconate. Eclampsia: grand mal seizure β give magnesium, protect from injury, O2, notify provider immediately. Delivery is the only cure.
Preeclampsia
BP β₯140/90 + proteinuria >20 wks
Severe
BP β₯160/110, HELLP, visual changes
Mag sulfate
Seizure prophylaxis β monitor DTRs
Mag toxicity
Absent DTRs, RR <12 β calcium gluconate
Treatment
Delivery is the only cure
Newborn Assessment
Normal newborn: HR 120β160, RR 30β60, Temp 36.5β37.5Β°C. Acrocyanosis normal. Caput succedaneum vs cephalohematoma.
Newborn Assessment
Key normal newborn findings β and the abnormalities that require intervention
Normal vitals: HR 120β160 (tachycardia if >160, bradycardia if <100), RR 30β60 (normal is fast!), Temp 36.5β37.5Β°C (axillary). Acrocyanosis: blue hands/feet normal for first few hours (peripheral circulation immature) β central cyanosis (lips, trunk) = abnormal. Caput succedaneum: edema crossing suture lines, present at birth, resolves in days. Cephalohematoma: bleeding under periosteum, does NOT cross suture lines, appears 24β48 hrs, resolves in weeks (risk for jaundice). Vernix (white coating), lanugo (fine hair), milia (white dots on nose) = all normal. Meconium: first stool within 24β48 hrs. Void within 24 hrs.
HR
120β160 bpm normal
RR
30β60 β fast is normal
Acrocyanosis
Blue hands/feet β normal
Caput
Crosses suture lines β resolves fast
Cephalohematoma
Does NOT cross suture lines β jaundice risk
Breastfeeding
Breastfeeding: latch = areola in mouth, not just nipple. Feed 8β12 times/24 hrs. Signs of adequate feeding: 6+ wet diapers/day by day 4.
Breastfeeding Support
NCLEX-tested breastfeeding education β latch, frequency, and signs of adequate intake
Good latch: baby's mouth covers areola (not just nipple), lips flanged outward, chin touching breast, audible swallowing, no pain. Feed on demand β 8β12 times per 24 hours (every 2β3 hrs). Duration: 10β15 min per breast. Signs of adequate intake: 6+ wet diapers/day by day 4, weight regain by day 10β14 (lose up to 10% initially), yellow seedy stools by day 4. Colostrum: first 3β5 days β high in antibodies (IgA), thick yellow, small amounts normal. Engorgement: frequent feeding, warm compress before feeding, cold compress after, supportive bra. Mastitis: breast infection β continue breastfeeding, antibiotics, warm compress, rest.
C-Section Nursing Care
Post C-section: assess uterine fundus, incision, lochia, pain, Foley output. Ambulate early (12β24 hrs) to prevent DVT.
Cesarean Section Nursing
Post-operative care after C-section β combining OB and surgical nursing care
Immediate post-op: assess as for any surgical patient PLUS obstetric assessments. Fundus: firm, midline, at umbilicus. Lochia: rubra (red, first 3 days), serosa (pink, days 4β10), alba (white, days 11+). Incision: Pfannenstiel (bikini line β horizontal). Foley catheter: usually removed 12β24 hrs post-op. Pain: multimodal analgesia β IV opioids β oral NSAIDS + acetaminophen β wean opioids. Early ambulation: 12β24 hrs β prevents DVT, ileus, pneumonia. Sequential compression devices (SCDs) until ambulating. Patient teaching: no driving for 4β6 weeks, lift nothing heavier than baby, incision care. Next delivery: VBAC possible for some.
Gestational Diabetes
GDM: diabetes diagnosed during pregnancy. Risks: macrosomia, hypoglycemia in newborn, shoulder dystocia, Cesarean delivery.
Gestational Diabetes
The metabolic complication of pregnancy β risks for mother and baby both tested on NCLEX
GDM: glucose intolerance first recognized during pregnancy. Screening: 24β28 weeks (1-hr glucose challenge test). Diagnosis: 3-hr OGTT. Pathophysiology: placental hormones β insulin resistance. Risks to baby: macrosomia (large baby β difficult delivery, shoulder dystocia), neonatal hypoglycemia (baby was compensating for mom's high glucose β baby's insulin remains high after birth β check newborn glucose at 1 hr). Risks to mother: UTIs, preeclampsia, C-section, future Type 2 DM (50% risk). Management: diet first (complex carbs, small meals), exercise, insulin if needed (NOT oral antidiabetics in pregnancy). Resolves after delivery.
Macrosomia
Large baby β shoulder dystocia risk
Newborn hypoglycemia
Check glucose at 1 hr
Management
Diet β exercise β insulin
Future risk
50% develop Type 2 DM
Newborn Jaundice
Physiologic jaundice: appears day 2β3, resolves by day 7β10. Pathologic: appears <24 hrs = EMERGENCY. Treatment: phototherapy.
Newborn Jaundice
The most common newborn condition β distinguishing physiologic from pathologic is key
Jaundice: yellow skin from bilirubin (RBC breakdown). Physiologic (normal): appears day 2β3 (after 24 hrs), peaks day 3β5, resolves day 7β10. Cause: immature liver, polycythemia. Pathologic (abnormal): appears within first 24 hours β Rh or ABO incompatibility, infection. EMERGENCY β needs immediate treatment. Phototherapy (bili lights): converts bilirubin to water-soluble form excreted in urine/stool. Nursing: eye shields (protect from light), turn every 2 hrs, increase feeds (hydration promotes excretion), monitor skin color and bilirubin levels, remove briefly for feeds. Kernicterus: bilirubin deposits in brain β permanent neurological damage.
Physiologic
Day 2β3, resolves day 7β10 β normal
Pathologic
<24 hrs β EMERGENCY, incompatibility
Phototherapy
Eye shields, turn q2h, increase feeds
Kernicterus
Bilirubin in brain β brain damage
Contraception
Most effective: implant > IUD > sterilization. Least effective: spermicide. Estrogen contraindicated: smokers >35, HTN, DVT history.
Contraception Nursing
Effectiveness rates and contraindications β the NCLEX expects nurses to counsel appropriately
Effectiveness (best to least): Implant (Nexplanon, >99%), IUD (Mirena/Paraguard, >99%), Sterilization (>99%), Depo-Provera shot (94%), Combined pill (91% typical use), Patch/Ring, Male condom (85% typical), Diaphragm (83% typical), Spermicide (72% typical). Estrogen-containing contraceptives CONTRAINDICATED in: smokers β₯35 years (DVT/PE risk), uncontrolled HTN, history of DVT/PE/stroke, breast cancer, migraines with aura, breastfeeding <6 weeks. Progestin-only (mini-pill, Depo, Mirena, Nexplanon): safer alternatives. Copper IUD (Paraguard): no hormones, also emergency contraception within 5 days.