🫀 Nursing · Medical-Surgical

Nursing tricks that make med-surg stick

Cardiac, respiratory, neuro, renal, and GI nursing — signs, symptoms, and interventions memorized.

🫀 Medical-Surgical

Memory tricks

Proven mnemonics — fast to learn, hard to forget.

MI — MONA
MI treatment: MONA — Morphine, Oxygen, Nitrates, Aspirin. 12-lead ECG within 10 min. Troponin is gold standard.
Myocardial Infarction
Recognizing and responding to MI — time is muscle, every minute counts
Classic MI symptoms: crushing chest pain (may radiate to jaw, left arm, back), diaphoresis, nausea, shortness of breath. Women/diabetics: atypical — jaw pain, fatigue, nausea only. MONA: Morphine (reduces preload/pain — now questioned in STEMI), Oxygen (if SpO2 <90%), Nitrates (vasodilation — hold if systolic <90 or recent sildenafil use), Aspirin (antiplatelet — 325 mg chewed). ECG: within 10 minutes of arrival. ST elevation = STEMI — needs cath lab within 90 min. Troponin: gold standard biomarker, rises 3–4 hr after MI. Give nothing by mouth (NPO) — may need procedure.
M
Morphine — pain/anxiety
O
Oxygen — if SpO2 <90%
N
Nitrates — vasodilate, hold if BP low
A
Aspirin 325 mg chewed
Stroke — FAST
Stroke: FAST — Face drooping, Arm weakness, Speech difficulty, Time to call 911. tPA within 3–4.5 hours of onset.
Stroke Recognition and Care
Time-critical emergency — recognizing stroke and the nursing response
FAST: Face drooping (ask to smile — asymmetry), Arm weakness (raise both arms — one drifts down), Speech difficulty (slurred or unable to speak), Time — call 911 immediately. Two types: Ischemic (87% — clot) and Hemorrhagic (13% — bleed). Treatment: Ischemic → tPA (alteplase) if within 3–4.5 hours of LAST KNOWN WELL, no hemorrhage on CT. Hemorrhagic → no tPA, manage BP, possible surgery. Nursing: NIH Stroke Scale assessment, position HOB 30°, NPO until swallow evaluation, falls precautions, BP management (allow permissive hypertension in ischemic unless giving tPA). Time is brain — 1.9 million neurons lost per minute.
F
Face drooping
A
Arm weakness
S
Speech difficulty
T
Time — call 911 now
tPA window
3–4.5 hours from last known well
Increased Intracranial Pressure
ICP signs: Cushing's Triad — Bradycardia, Hypertension (widening pulse pressure), Irregular respirations. LATE sign = impending herniation.
Increased ICP
Cushing's Triad is a medical emergency — the nurse must recognize it and act immediately
Normal ICP: 5–15 mmHg. Early ICP signs: headache (worse with straining), nausea/vomiting (projectile), altered LOC, pupil changes (unequal, sluggish). Late sign — Cushing's Triad (EMERGENCY): Bradycardia + Hypertension (widening pulse pressure) + Irregular/slow respirations → impending brainstem herniation. Nursing: HOB 30°, head midline (no neck rotation — impairs venous drainage), avoid clustering care, dim lights/quiet environment, avoid Valsalva (no straining), monitor pupil response. Do NOT: suction vigorously, hip flexion >90°, prone positioning.
Early
Headache, N/V, altered LOC
Cushing's Triad
Bradycardia + HTN + irregular RR
Position
HOB 30°, head midline
Avoid
Straining, Valsalva, clustering care
Respiratory — COPD vs Asthma
COPD: chronic, progressive, barrel chest, pursed-lip breathing. Asthma: episodic, reversible, wheezing, triggered. O2 cautiously in COPD.
COPD vs Asthma
Two obstructive lung diseases with important differences — NCLEX loves the oxygen question in COPD
COPD (emphysema + chronic bronchitis): irreversible airway obstruction. Emphysema: barrel chest, pursed-lip breathing, decreased breath sounds, 'pink puffer' (fights to breathe). Chronic bronchitis: productive cough >3 months/2 years, 'blue bloater.' O2 in COPD: hypoxic drive — give O2 2–3 L/NC, target SpO2 88–92% (not 95–100%). High O2 may suppress respiratory drive. Asthma: reversible bronchospasm, triggered (allergens, exercise, cold). Wheezing on expiration. Peak flow meter: green >80%, yellow 50–80%, red <50%. Rescue inhaler (albuterol) before preventive (corticosteroid inhaler).
COPD O2
2–3 L, target SpO2 88–92%
Emphysema
Barrel chest, pursed lips, pink puffer
Chronic Bronchitis
Productive cough, blue bloater
Asthma
Reversible, wheezing, rescue before preventive
Pneumonia
Pneumonia assessment: fever, productive cough, crackles, decreased breath sounds. Position: semi-Fowler's. Encourage fluids and deep breathing.
Pneumonia Nursing
The most common hospital-acquired infection — assessment, positioning, and prevention
Signs: fever and chills, productive cough (yellow/green/rust-colored sputum), pleuritic chest pain (worse with breathing), crackles and decreased breath sounds in affected lobe, tachypnea, hypoxia. Community-acquired (CAP): S. pneumoniae most common. Hospital-acquired (HAP): gram-negative organisms, MRSA. Nursing care: semi-Fowler's position (HOB 30–45°), encourage fluids (2–3 L/day unless restricted — thins secretions), deep breathing and coughing exercises, incentive spirometer, turn every 2 hours, ambulate early. Prevention: pneumococcal vaccine, hand hygiene, oral care in ventilated patients (VAP bundle).
Diabetes — Hypo vs Hyperglycemia
Hypoglycemia (<70): Cold and Clammy = give candy. Hyperglycemia (>180): Hot and Dry = sugar high.
Hypoglycemia vs Hyperglycemia
The quick way to distinguish and treat two dangerous blood sugar extremes
Hypoglycemia (<70 mg/dL): Cold and Clammy — diaphoresis, tremors, tachycardia, confusion, seizure. Cause: too much insulin, missed meal, excess exercise. Treatment: 15-15 rule — 15g fast carbs (4 oz juice, glucose tablets), recheck in 15 min. If unconscious: IV dextrose (D50) or glucagon IM. Hyperglycemia (>180–250): Hot and Dry — polyuria (3 Ps: Polyuria, Polydipsia, Polyphagia), fruity breath (DKA), Kussmaul respirations (deep, rapid — blowing off CO2 in DKA). DKA (Type 1): ketones, pH <7.3. HHS (Type 2): extreme hyperglycemia, no ketones, elderly. Treatment: insulin drip, IV fluids, K+ replacement.
Hypo <70
Cold/clammy, diaphoresis, tremor, confusion
Hypo Tx
15g carbs → recheck 15 min, or IV D50
Hyper
Hot/dry, 3 Ps, fruity breath
DKA
Ketones, Kussmaul breathing, pH <7.3
Renal Failure — AEIOU
AEIOU — Acute kidney injury complications: Acidosis, Electrolyte imbalances (K+↑), Intoxication (uremia), Overload (fluid), Uremia.
Acute Kidney Injury
Recognizing and managing AKI — the NCLEX expects nurses to monitor and intervene
AKI: rapid decline in kidney function over hours to days. Stages (RIFLE/KDIGO): Risk, Injury, Failure, Loss, ESKD. Oliguric phase: urine output <0.5 mL/kg/hr, BUN and creatinine rise, K+ rises (hyperkalemia — most dangerous). AEIOU complications: Acidosis (metabolic), Electrolyte imbalance (hyperkalemia → EKG changes, peaked T-waves → cardiac arrest), Intoxication (uremia — confusion, asterixis), Overload (fluid), Uremia (N/V, pericarditis, pruritus). Nursing: strict I&O, daily weights, low K+ diet, BP monitoring, dialysis access care. Fluid challenge: 500 mL NS bolus if pre-renal cause.
A
Acidosis — metabolic
E
Electrolytes — hyperkalemia, peaked T waves
I
Intoxication — uremia, confusion
O
Overload — fluid
U
Uremia — N/V, pericarditis, pruritus
Postoperative Care
Post-op ABCDE: Airway, Breathing, Circulation, Drugs (anesthesia), Everything else (pain, N/V, wound).
Postoperative Nursing
Systematic assessment immediately after surgery — and the complications to watch for
Immediate post-op (PACU): Airway — maintain, suction if needed. Breathing — respiratory rate, SpO2, breath sounds. Circulation — BP, HR, bleeding at surgical site. Drugs — anesthesia reversal, pain management. Temperature — hypothermia common (warm blankets, forced air). Early complications: respiratory depression (opioids — give Narcan), airway obstruction (tongue, secretions), hemorrhage (increasing HR, decreasing BP), emergence delirium. Late complications: atelectasis (encourage deep breathing, IS), DVT (SCDs, early ambulation, anticoagulants), wound infection (3–5 days post-op fever), paralytic ileus (listen for bowel sounds).
Fluid and Electrolytes — Big 5
Hypokalemia: U waves, weak muscles. Hyperkalemia: peaked T waves. Hyponatremia: confusion, seizures. Hypernatremia: thirst, dry mucosa.
Electrolyte Imbalances
The five electrolytes NCLEX tests most — know the critical values and EKG changes
Potassium (normal 3.5–5.0): Hypo (<3.5): muscle weakness, cramps, U waves on EKG, constipation. Causes: diuretics, vomiting, NG suction. Replace slowly (never IV push — fatal). Hyper (>5.5): peaked T waves, wide QRS, muscle weakness, cardiac arrest. Treat: calcium gluconate (protect heart), insulin+dextrose (shift K+ into cells), Kayexalate. Sodium (normal 135–145): Hypo (<135): headache, confusion, seizures — restrict fluids, hypertonic saline (slowly or central herniation). Hyper (>145): thirst, dry mucosa, restlessness, seizures — free water replacement. Calcium: Hypo — Trousseau's and Chvostek's signs, tetany. Magnesium: Hypo — cardiac dysrhythmias.
Hypokalemia
U waves, muscle weak — diuretics cause
Hyperkalemia
Peaked T waves — cardiac emergency
Hyponatremia
Confusion, seizures — fluid restrict
Hypernatremia
Thirst, dry — give free water slowly
Wound Care and Pressure Injuries
Pressure injury stages: I (redness), II (partial thickness), III (full thickness), IV (bone/tendon visible). Turn every 2 hours.
Pressure Injuries
Staging wounds and preventing pressure injuries — prevention is always better than treatment
Stage I: intact skin, non-blanchable redness. Intervention: relieve pressure, moisturize. Stage II: partial thickness skin loss — shallow open ulcer or blister. Stage III: full thickness skin loss, subcutaneous tissue visible, no bone/tendon. Stage IV: full thickness, bone/tendon/muscle exposed. Unstageable: covered by eschar — cannot stage until debrided. Deep tissue injury (DTI): purple/maroon discoloration, intact skin. Prevention: turn every 2 hours, pressure-relieving mattress, keep dry (moisture = skin breakdown), adequate nutrition (protein + vitamin C + zinc), assess Braden scale. Never massage over bony prominences — increases breakdown.
Stage I
Non-blanchable redness — intact skin
Stage II
Blister or shallow ulcer
Stage III
Full thickness — no bone visible
Stage IV
Bone/tendon visible
Prevention
Turn q2h, Braden scale, nutrition
ABG Interpretation
ABGs: pH 7.35–7.45, PaCO2 35–45, HCO3 22–26. ROME: Respiratory Opposite, Metabolic Equal.
ABG Interpretation
The step-by-step method for reading arterial blood gases — ROME makes it systematic
Normal values: pH 7.35–7.45, PaCO2 35–45 mmHg (respiratory), HCO3 22–26 mEq/L (metabolic). Step 1: pH — acidosis (<7.35) or alkalosis (>7.45)? Step 2: PaCO2 — if it matches pH direction (opposite), it's respiratory. Step 3: HCO3 — if it matches pH direction (same), it's metabolic. ROME: Respiratory Opposite (pH up, CO2 down = alkalosis), Metabolic Equal (pH up, HCO3 up = alkalosis). Compensation: the system NOT causing the problem tries to correct pH. Respiratory acidosis (hypoventilation, COPD): pH↓, CO2↑. Metabolic acidosis (DKA, renal failure): pH↓, HCO3↓. Metabolic alkalosis (vomiting, NG suction): pH↑, HCO3↑.
pH <7.35
Acidosis
pH >7.45
Alkalosis
CO2 matches pH?
Respiratory cause
HCO3 matches pH?
Metabolic cause
ROME
Respiratory Opposite, Metabolic Equal